Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Alternatively, our capacity to rescue SERT function in KI midbrain synaptoneurosomes through the inhibition of FAK suggests elevated FAK signaling downstream of your Pro32Pro33 mutant, as confirmed by greater pFAK localization in five-HT synapses. https://pro33login11000.blogdal.com/32695678/an-unbiased-view-of-pro33-login